Why Formula Milk Should be Illegal

In Short :
Newly born mammals are relatively helpless. To ensure optimum development of the baby, mother's milk contains different substances enhancing growth and development of organs. Mother's milk contents differ very much from cow's milk contents (and soy milk contents of course). Logically, the less human infants have been breast-fed, they generally become dumber (1):  

* "breastfeeding associated with significantly higher scores for cognitive development than formula feeding"

* "duration of breastfeeding significant predictor of later cognitive or educational outcomes"

* "the longer the child has been breastfed, the more pronounced the effect"

* "prolonged breastfeeding significantly related to scores reflecting general cognitive capacity"

* "the exclusively breastfed group showed more advanced"

* "beneficial effect of breastfeeding on postnatal neurological development"

* "breastfeeding associated with higher mean in children's intelligence"

* "more infants fed term formula had low verbal IQ"

* "significant association between duration of breast milk feeding and verbal IQ"

* "strong positive relationship between breastfeeding and PPVT-R scores" 


...and fatter (2), and more susceptible to infections and diseases. (3)

Every child deserves the best start in life, and therefore has to be breast-fed for at least 1½ year. If your child is born with teeth, or gets these within half a year, and feeding hurts too much, check La Leche League (FAQ on biting) or you can use a pump.

Many foods contain chemicals that interfere with hormones that regulate lactation. To prevent the child from rejecting mother's milk *and to prevent allergies), mama needs to adapt her diet. 


Mama also benefits from breast-feeding her child : breast-feeding = losing adipose fat. When she breastfeeds, the body produces different hormones increasing transformation of adipose fat into mothers’ milk-fat. (supplying the child with 52% of required energy) 




 Hail to the American Acadamy for Pediatrics

for their excellent new policy






In Detail :


Why is this a picture you see far too little in pregnancy-magazines ?

Because breast-feeding does not increase any company’s profits.

And been fully weaned, one will less crave for junk food. 

There is no freedom of choice for humans
if it has been taken away from them
at the beginning.

Breast-feeding is not a choice,
but an obligation to the choice.

Give your child the freedom of choice.


All mammals are born too early, but because our brains are relatively big, human babies are born far too early; Human gestation length is 9 months instead of the expected 21 months, for a specie this size. To compensate the loss of gestation time, by nature human infants have to be breast-fed for at least 1½ years.

Logically, mother's milk of all mammals contains a precisely balanced cocktail of hormones, growth factors and other 'messenger'-peptides to stimulate optimum development of the young.

Some of the growth factors, hormones and peptides in human milk, cow's milk and milk from other mammals :



- bombesine(a neuropeptide) (4)

- GRP (Gastrin-releasing peptide) (\5)

- substance P (a neurotransmitter) (6)

- CGRP (calcitonin-gene-related peptide, also a neurotransmitter) (6)

- IGF-1 (Insulin-like growth factor-1) (7)

- IGF-2 (7)

- EGF (Epidermal growth factor) (8)

- NGF (Nerve growth factor) (9)

- PRP (Prolactin-releasing peptide (10)

- LHRH (or : GnRH, stimulates secretion of LH and FSH) (11)

- progesterone (12)

- peptide YY (13)

- peptide histidine methionine (13)

- neuropeptide Y (stimulating appetite) (14)

- TRH (stimulating TSH secretion) (15)  TRH stimulates prolactin- (16) and GH secretion (17), through T3. (189

- TSH (stimulating T3- and T4 secretion) (18)

- T3 (Triiodothyronine) (20) T3 increases the number of estrogenreceptors, increasing estrogen-influence. (21)

- GHRF (Growth-hormone-releasing factor) (22) GHRF stimulates GH- and (through GH) IGF-1 and –2 secretion (23)

- ACTH (regulating cortisol secretion) (22)

- neurotensine (24)

- cortisol (25)

- insuline (regulating blood-glucose level) (26)

- beta-endorphine (opioid peptide) (27)

- small opioid peptides (see site12)

- benzodiazepine-agonist peptides (neurotransmitters) (28).



Compared to other mammals, humans need very different amounts of nutrients, hormones and other 'messenger'-substances. And logically, mothers' milk contents of all different mammals differs very much.

Because formula milk is based on cow's milk, or soy-protein, formula milk never ever contains the right nutrients or 'messenger' substances.

Furthermore, babies fed with cow’s-milk-based formula milk already become addicted to cow’s milk-opioid peptides in their very first stage of life, guaranteeing increased cravings for food products containing milk protein in adult life, thus enhancing obesity. Of course many food products contain cow’s milk protein, to increase sales. (check labels).


Soy-based formula milk is even worse than cow’s milk-based formula milk ; like soy, soy-based formula milk contains phyto-estrogens (29) disturbing hormone levels (30). Some phyto-estrogens are mutagenic (31) and can enhance brain tumors (32). (brain cancer is the main type of cancer in infants) For more info about the down-sides of soy, see this site and external info.


Logically, formula-fed babies do not get all the substances they need for optimum brain development.

In the United States only 20% of babies are breastfed at six months, and in the UK only 25% of babies are breastfed at four months, which is one of the reasons why people increasingly become more fat, dumb and sick.





But What If You Cannot Give milk?

Though you couldn’t have known, very probably you are to blame.

To make sure you will produce sufficient milk and your baby will not reject it, you have to consume extremely little prepared food and no dairy- or wheat-products when pregnant, as well as before, and when lactating. (see “Maternal Food” and “Maternal Food & Breastfeeding”)

Your baby may reject your milk simply because it contains too much bad substances (a natural reaction)

Especially consuming milk yourself while pregnant or afterwards, inhibits your own production of milk. This is because cow's milk partially contains the same hormones as your body secretes while lactating; so that these hormones from consumed milk have a negative feedback on the secretion of these hormones by your body, and thus on the production of human milk in your body. Consuming milk kind of mimics the situation of when lactating. (that is why women averagely have bigger breasts nowadays)




But what if I cannot sufficiently reverse that anymore?


Breastfeeding in combination with instant milk is NOT an option; your child will have digestive problems and cramps, and will vomit and cry a lot.

But there is a very simple and effective solution, not compromising the child’s interests : Let your child drink the milk from another woman (she can use a pump) ; most women can produce milk for two, while having only one child. Yes that is unusual, and will cost you some, but your child is worth it. And yes, of course that other women has to be tested on all possible diseases.





Mother’s Milk Versus Formula Milk

Unquestionable, human milk is designed to support the development of large brains, capable of processing and storing lots of information. Milk from mammals like cows isn’t, but is however designed to support other functions, like constant grazing.



Of course, when formula milk is composed of soymilk, formula milk does not contain any of the essential 'messenger'-substances. When composed of cow's milk, at best its contents support optimum development of the calf, not human young. ‘Unfortunately’, formula milk can not be composed of raw cow’s milk protein. And due to the influence of the heat involved in the preparation process, both messenger substances are destroyed and dangerous HCA are formed.

Even at best, formula milk lacks the most important messenger-substances (33).


For example, when composed of cow's milk, formula milk does not contain CIF (colostrum inhibiting factor) ; mother's milk does, but cow's milk doesn't (34). And in (cow's-) formula milk, EGF level is far lower, while EGF protects the infant's colon. (35) Spermine and spermidine are important factors in metabolic and immunologic processes, but level of these amines in formula milk is 10 times lower as in mother's milk. (36)




Regarding essential messenger substances, formula milk for 3 days old babies is no different than formula milk for 3 months old infants. How could we ever think any formula milk can compete with mother’s milk any way ? ; Mama produces milk that ingeniously is different every single day !!!! 


In general, mother’s milk neuropeptide levels decrease after 6 weeks, except for the GIP level, which decreases only after 36 weeks. (37) In 1st day post partum mother's milk IGF-1 level is 5 times higher than IGF-2 level. From day 3 to 7 IGF-1 level decreases 80% and IGF-2 level increases 200%. And from day 1 to 7 the IGFB-2 level increases 1900% !!! (38) In comparison to the average full-term mother's milk, cow's milk IGF-1 level is 3 to 4 times lower.

Formula milk does not even contain any IGFs at all !!! (39)




Mama's milk is also carefully adapted to the changing needs of her baby. Formula milk manufacturers don't even try to imitate mama’s product ; because they never can. (though of course they claim to) And even if they could come close ; requirements of every child differ, and only mama's body 'knows' what her baby needs, because she already nourished her baby before it was born.

Logically, blood-peptide levels in children receiving formula milk differ very much from those in children receiving human milk ;


In children receiving human milk, blood-insulin-, -GIP- (gastrin-inhibiting peptide), -PPP- (pancreatic polypeptide), and -CCK-level (cholecystokinine) is lower, and blood-gastrin level is higher. (40)





Mother's milk contents vary greatly, but even compared to the maximum and minimum levels, average formula milk contains far too much (or too little) of almost every nutrient.

Most seriously, formula milk contains an average of 10 to 23 times as much iron as the maximum in mother's milk. Excess amounts of metals like iron is pro-oxidative (41), damaging nutrients, arteries (42) messenger-substances, cell-DNA (43) and enzymes (44), increases heart attack risk and can cause diabetes (45), colon-cancer (46) Parkinson's disease (47) and infertility. (48)  

Formula milk averagely contains 21 to 28 times as much vitamin D as mother’s milk maximally does. Excessive vitamin D causes arthritis and arteriosclerosis. 

Also, formula milk contains twice as much calcium and 3 times as much phosphorus, causing both arteriosclerosis and bone-decalcification


The reason why they put so much metals in formula milk is because the bioavailability of iron, zinc, copper, manganese, selenium and vitamin B9 (folic acid) in cow's and formula milk is much lower. (49) Why ? Because cow’s milk and soy contain different substances not meant for the human body. (also see site3) For example : soy-based formula milk contains phyto-estrogens, which easily bind to iodide, causing iodide deficiencies. (50)


The whole idea behind formula milk is to put in far too much of certain nutrients because you never know how much is absorbed. Formula milk manufacturers apparently like to gamble, with your child’s health.




Humans need relatively little protein, because our large brains need lots of sugars ; we daily need 125-150 gram of pure glucose for the brain only. Logically, our primary food, mother’s milk, contains relatively little protein, because extra vitamin B2, B6 and folic acid is required to process excessive protein.

Cow's milk contains 220% more protein than human milk. Sheep milk contains 366%, horse milk 95% and goat's milk 227% more protein than human milk.

Also, protein-composition differs very much ; Casein is a relatively indigestible protein, containing opioid peptides. Cow's milk contains 7 times as much casein, sheep milk 12 times, horse milk 3 times and goat's milk 8 times as much casein as in mothers' milk. (51)

Mother's milk protein composition is of course designed to the need of human suckling. Logically, in relation to premature infants’ need for essential amino acids, mother's milk protein composition is 35% better than cow’s milk (3,5%-fat) and raw soy protein. (see bottom site18) Soy however, also requires different preparations before ending up in formula milk, and the heat involved destroys amino acids (and originates mutagenic HCA). That's why formula milk contains 24 to 95% more protein, containing however more amino acids (or other nitrogen-compounds) babies cannot use for construction purposes. Processing extra useless protein does require extra vitamin B2, B6 and folic acid, inhibiting growth.




The human brain alone needs 125 to 150 gram of pure glucose a day. The brains of other mammals are relatively smaller, and they therefore need relatively less sugars.

Logically, human milk contains most sugars ;

Human milk contains 54% more sugars than cow's milk and sheep milk, 13% more than horse milk, and 67% more than goat's milk. (51) Logically, breastfed babies are much happier, don’t cry much and sleep a lot.




Equally important to the brain is cholesterol. Cholesterol is one of the substances making sure your baby sleeps well. And no, undamaged cholesterol from raw animal food is not bad at all. 

Logically, human milk contains most cholesterol ; 103% more than cow's milk, and 127% more than goat's milk. (51)



Fatty acids

Long chain polyunsaturated fatty acids are essential to the development of the brain. One such an essential fatty acid is DHA. DHA levels are significantly higher in exclusively breastfed children. (78) Higher DHA levels are associated with better sleeping babies and greater central nervous system maturity. (79)




Maternal Food

What the pregnant mother consumes, greatly influences her baby. Like pregnant mothers should not consume cigarettes, drugs or alcohol. And because prepared food contains partly the same chemicals as cigarettes do, and because some of these chemicals elicit equal effects as those in drugs, it is extremely important what foods pregnant women consume. Of course most people don’t want to know this, because it takes much discipline to do what is right when pregnant. But in fact it is very simple, if you are not ready to do everything what is right for your baby, it is not (the) right (time) for you to have a baby ;


-            Pregnant women consuming prepared meat increase baby’s brain-cancer risk. (52) Though prepared meat contains most mutagenic heterocyclic amines, other prepared foods contain such chemicals too. (proteinacous prepared foods in particular)

-            Pregnant women consuming foods containing trans-fatty acids, elevate trans-fatty acid level in mother's milk (53). Trans fatty acids are unnatural fatty acids that also originate due to the influence of heat. Maternal trans-fatty acid consumption increases vascular diseases- (54), breast cancer- (55) and pre-eclampsia risk. (56)

-      Because many lactating women drink cow's milk (and did drink it before), in 60% of suckling babies the immune system reacts upon cow's milk-lacto globulin in mother's milk. (57)



To prevent mother's milk from being rejected by the baby, women should not consume any dairy products while pregnant or lactating, as well as during pre-pregnancy. To prevent the child being born addicted to wheat-opioids, the mother should also not consume any wheat products. And to prevent the child from getting brain-cancer etc., mama should consume as little prepared food as possible. She should consume as much fruit and other brainfood, containing all the nutrients she and her baby need. Consuming lots of fruits, she doesn't need supplementary folic acid, because then she consumes sufficient, but not excessive protein

Though consuming protein requires extra vitamin B9 (folic acid), mamas-to-be are still advised to consume more protein, causing folic acid deficiencies. Logically, maternal folic acid supplementation doesn't really work (58) ; the cause remains !!





Maternal Food and Breastfeeding

Being able to breastfeed has nothing to do with big or small breasts. What really influences your ability to breastfeed, is the food you consume when lactating, pregnant, and before. Most women do not want to believe it, but the bad foods pregnant women consume, can diminish their capacity to breastfeed.

How come?

Breastfeeding strongly influences hormone metabolism, and vice versa.

Like all women know, we can influence our hormone metabolism through absorbing exogenous hormones (the pill). Unfortunately, also chemicals in food can impair our hormone metabolism. Phyto-estrogens in soy, for example, can even cause infertility.


There are a few hormones regulating composition of mother’s milk. (see “Breastfeeding & Losing weight” below) Three of these hormones are:

-    Oxytocin

-    Leptin

-    Prolactin



Unfortunately, prepared foods contain beta-carbolines and wheat- and dairy products contain opioid peptides.

Beta-carbolines can impair oxytocin- (through GABA-receptors) (59), CRF- (leptin acts through CRF) (60) and prolactin-metabolism (through reducing the secretion of the thyroid hormone TSH) (61)

Opioid peptides can also impair oxytocin- (62) (even if the mother breast-feeds her baby (63)) and CRF-metabolism. (64)

What does this mean?

That food-chemicals can impair your ability to breastfeed.




Breastfeeding & Losing weight

After having given birth, mama needs to lose weight, to be able 'to flee from danger'. The baby on the other hand, needs to gain weight. Nature therefore did a little math and came up with the perfect solution ; no less than an average 52,5% of energy required by the baby is supplied by mama's fat in mothers’ milk. To make this happen, secretion of a few hormones is increased by breastfeeding, making mama thinner ;


Oxytocin inhibits appetite. (65) (and enhances post-natal uterus shrinking (66)) By giving birth (and clitoral orgasm), lots of oxytocin is released. Anesthesia however, inhibits this release of oxytocin. Luckily, every time and as long as the mother breast-feeds her baby, oxytocin is released too. (67) But if you stop giving mother's milk, oxytocin release decreases, and appetite increases.

Opioid substances (from dairy- or wheat products or morphine) inhibit oxytocin release, even when breast-feeding. (68)


Leptin ; Of course women who delivered a baby contain more adipose fat, which stimulates leptin production. Leptin is produced in adipose tissue, and signals at different neuropeptides and hormones to inhibit appetite. (69) The release of leptin is however inhibited when mama does not breast-feed her baby. (70)


Prolactin ; As long the mother is breast-feeding her baby, production of yet another hormone, prolactin, is increased. Prolactin inhibits appetite and enhances transformation of adipose fat into mother's milk-fat and / or available energy (71)



Logically, women who breast-feed their babies, preferably for at least 1½ year, much easier (re)gain their set-point weight. (72) Secretion of oxytocin, leptin and prolactin, however, is impaired by beta-carbolines and opioid peptides. Which means that consuming as little proteinous prepared food and no wheat- or dairy products, it is even easier to lose excess body-fat.


When taking care of multiple small infants in nature, it is extremely difficult to flee from danger. Therefore the female body prevents nursing women from becoming pregnant again ; breast-feeding inhibits secretion of hormones stimulating ovulation (GnRH, LH and FSH) (73). And that works ; birth intervals are longer in women who exclusively breast-fed their babies. (74) (do use a condom though)



Of course all of you wondered “How do they do that ?” “How come all these soapstars, moviestars and models get so thin, so fast after they popped their little ones ?” Well, first of all, they don’t eat for two, they don’t drink milk, consume wheat products or other ‘normal-people’s-food’. And because of that they produce sufficient milk to feed their babies. And they fully nurse their children. That’s why they look fabulous, with gorgeous hair and they even look thinner than ever before. So now you know.



So breastfeeding strongly influences maternal hormone metabolism, regulating hormonal changes due to the pregnancy. There is no doubt one has to come after the other. If not, pregnancy-hormone cycle has not been completed, and post-natal hormone metabolism is impaired, causing post-natal depressions etc. The uterus of non-breastfeeding women for example, does not shrink back to its pre-pregnant size anymore. (66) Logically, breast- (75) ovarian (76) and endometrial (77) cancer risk in women who did not breastfeed their baby, is increased.







For more info about what the formula milk industry is doing, please visit Why Boycott Nestle / Risks of Artificial Feeding



For shocking info about vaccinations, check out Think Twice


For State-vaccine laws, check out New-Atlantean


To get in contact with other visitors regarding vaccinations, visit Odomnet






Kid's - food

When infants can begin to eat solid food, it is extremely important they are supplied with the right foods.

Prepared food causes ADHD, obesity, cancer and brain-diseases, and inhibits cognitive functions. Dairy- and wheat products cause allergies, numbness and obesity. (see index)

Fruit and raw animal food are easy to digest, which enhances growth, and they contain all nutrients infants need. (see site3) In combination with fruits, fresh raw salmon is best alternated with fresh raw biological egg yolk (and –tuna).


There is nothing wrong with small children consuming fresh raw salmon. Consuming fresh raw foods strengthens the child's defense system. Raw salmon and -egg yolk are very nutritious and easy to digest. Be however very sure to buy really fresh (and biological) salmon and eggs. 

Beware: If your child is not used to consuming raw egg yolk or –salmon, begin very carefully. The child needs time and small quantities of raw food to empower its defense system in reaction to small amounts of bacteria. For the first week, give your child only half a teaspoon fresh raw egg yolk or one bite of fresh raw salmon a day.


Of course it is always very tempting for food-retailers to sell not-so-fresh-anymore fish, instead of throwing it away. And very often the fish has been frozen some time. Raw fish can best be purchased in Japanese restaurants, or where they buy their fish (because of their sushi-tradition).

Be sure to buy eggs that originate from chickens that have been fed raw grains; to enlarge profits, most animals are fed recycled scraps and wastes, very often containing animal residues, containing bad –fats, -cholesterol and –protein and toxins.

Unfortunately, most salmon (and all other animals) are fed the same trash. Be extremely investigative and picky.

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(1) Horwood LJ, et al, Breast milk feeding and cognitive ability at 7-8 years. Arch Dis Child Fetal Neonatal Ed 2001 Jan;84(1):F23-7. , Quinn PJ, et al, The effect of breastfeeding on child development at 5 years: a cohort study. J Paediatr Child Health 2001 Oct;37(5):465-9. , Anderson, J.W. et al, Breast-feeding and cognitive development: a meta-analysis. Am. J. clin. Nutr. 1999 / 70 (4) / 525-535. , Morris, B.H. et al, Feeding, medical factors, and developmental outcome in premature infants. Clin. Pediatr. (Phila) 1999 / 38 (8) / 451-457. , Horwood, L.J. et al, Breastfeeding and later cognitive and academic outcomes. Pediatrics 1998 / 101 (1) / E9. , Golding, J. et al, Association between breast feeding , child development and behaviour. Early Hum. Dev. 1997 / 49 / suppl.S175-184. , Niemela, A. et al, Is breastfeeding beneficial and maternal smoking harmful to cognitive development of children ? Acta. Paediatr. 1996 / 85 (10) / 1202-1206. , Johnson DL, et al, Breast feeding and children's intelligence. Psychol Rep 1996 Dec;79(3 Pt 2):1179-85. , Wang, Y.S. et al, The effect of exclusive breastfeeding on development and incidence of infection in infants. J. Huym. Lactation. 1996 / 12 (1) / 27-30. , Lanting, C.I. et al, Neurological differences between 9-year-old children fed breast-milk or formula-milk as babies. Lancet 1994 / 344 (8933) / 1319-1322. , Rogan, W.J. et al, Breast-feeding and cognitive development. Early Hum. Dev. 1993 / 31 (3) / 181-193. , Lucas, A., Breast milk and subsequent intelligence quotient in children born preterm. Lancet 1992 / 339 (8788) / 261-262. , Morrow-Tlucak, M. et al, Breastfeeding and cognitive development in the first 2 years of life. Soc. Sci. Med. 1988 / 26 (6) /635-639. , Fergusson, D.M. et al, Breast-feeding and cognitive development in the first seven years of life. Soc. Sci. Med. 1982 / 16 (19) / 1705-1708.  
(2) Agostoni, C., Breast feeding and childhood obesity. Pediatr. Res. 2000 / 47 (1) / 3. , Yadav, M. et al, Breast-feeding and childhood obesity. J. Pediatr. Gastroenterol. Nutr. 2000 / 30 (3) / 345-346. , Campbell, C. ,Childhood obesity. Breast feeding is important. Brit. Med. J. 2000 / 320 (7246) / 1401 / disc.1402-1403. , von Kries, R. et al, Breast feeding and obesity : cross sectional study. Brit. Med. J. 1999 / 319 (7203) / 147-150. Breast feeding seems to reduce the risk of obesity in children. (no authors listed) Brit. Med. J. 1999 / 319 (7203) / B. Xu, J.F., Case control study of child obesity. (in Chinese) Chung Hua Yu Fang I Hsueh Tsa Chih 1990 / 24 (3) / 146-148. , Neumann, G. et al, Breast feeding and early postnatal weight gain in Potsdam. (in German) Zentralbl. Gynakol. 1986 / 108 (7) / 411-418.
(3) Sperl, W. et al, Sudden Infant Death Syndrome prevention program in Tyrol. (in German) Wien Klin. Wochenschr. 2000 / 10 (112) / 209-215. , Virtanen, S.M. et al, Cow’s milk consumption, HLA-DQB1 genotype, and type 1 diabetes: a nested case control study of siblings of children with diabetes. Childhood diabetes in Finland study group. Diabetes 2000 / 49 (6) / 912-917. , Tsutsumi, O. et al, Breast-fed infants, possibly exposed to dioxins in milk, have unexpectedly low incidence of endometriosis in adult life. Int. J. Gynaecol. Obstet. 2000 / 68 (2) / 151-153. , Villalpando, S. et al, Growth faltering is prevented by breast-feeding in underpriviliged infants from Mexico City. J. Nutr. 2000 / 130 (3) / 546-552. , Lima, A.A. et al, Persistent diarrhea signals a critical period of increased diarrhea burdens and nutritional shortfalls: a prospective cohort study among children in northeastern Brazil. J. Infect. Dis. 2000 / 181 (5) / 1643-1651. , Smulevich, V.B. et al, Parental occupation and other factors and cancer risk in children: 1. Study methodology and non-occupational factors. Int. J. Cancer 1999 / 83 (6) / 712-717. , Shu, X.O. et al, Breast-feeding and risk of childhood acute leukemia. J. Natl. Acad. Cancer Inst. 1999 / 91 (20) / 1765-1772. , Silfverdal, S.A. et al, Protective effect of breastfeeding: an ecologic study of Haemophilus influenza meningitis and breastfeeding in a Swedish population. Int. J. Epidemiol. 1999 / 28 (1) / 152-156. , Perera, B.J. et al, The impact of breastfeeding practises on respiratory and diarrhoeal disease in infancy: a study from Sri Lanka. J. Trop. Pediatr. 1999 / 45 (2) / 115-118. , Long, K. et al, The impact of infant feeding patterns on infection and diarrheal disease due to enterotoxigenic Escheria coli. Salud. Publica Mex. 1999 / 41 (4) / 263-270. , Hypponen, E. et al, Infant feeding, early weight gain, and risk of type 1 diabetes. Childhood Diabetes in Finland (DiMe) Study Group. Diabetes Care 1999 / 22 (12) / 1961-1965. , Hokama, T. et al, Incidence of Haemophilus influenza in the throats of healthy infants with different feeding methods. Pediatr. Int. 1999 / 41 (3) / 277-280. , Hanson, L.A. ,Human milk and host defence:immediate and long-term effects. Acta Paediatr. Suppl. 1999 / 88 (430) / 42-46. , Davis, M.K., Revieuw of the evidence for an association between infant feeding and childhood cancer. Int. J. Cancer. Suppl. 1998 / 11 / 29-33. , Deb, S.K. ,Acute respiratory disease survey in Tripura in case of children below five years of age. J. Indian Med. Assoc. 1998 / 96 (4) / 111-116. , Virtanen, S.M. et al, Cow’s milk consumption, disease-associated autoantibodies and type 1 diabetes mellitus : a follow-up study in siblings of diabetic children. Childhood Diabetes in Finland Study Group. Diabet. Med. 1998 / 15 (9) / 730-738. , Corrao, G. et al, Risk of inflammatory bowel disease attributable to smoking, oral contraception and breastfeeding in Italy: a nationwide case-control study. Cooperative Investigators of the Italian Group for the study of the Colon and the Rectum (GISC). Int. J. Epidemiol. 1998 / 27 (3) / 397-404. , Langhendries, J.P. et al, Intestinal flora in the neonate: impact on morbidity and therapeutic perspectives. (in French) Arch. Pediatr. 1998 / 5 (6) / 644-653. , Bouguerra, F. et al, Breast feeding effect relative to age of onset of celiac disease. Arch. Pediatr. 1998 / 5 (6) / 621-626. , Golding, J. et al, Does breast feeding protect against non-gastric infections ? Early Hum. Dev. 1997 / 49 / suppl.S105-120. , Silfverdal, S.A. et al, Protective effect of breastfeeding on invasive Haemophilus influenza infection: a case-control study in Swedish preschool children. Int. J. Epidemiol. 1997 / 26 (2) / 443-450. , Covington, M.T. et al, Receipt of recommended prenatal interventions and birth weight among African-American women: analysis of data from the 1988 National Maternal and Infant Health Survey. Matern. Child. Health J. 1997 / 1 (3) / 157-164. , Nafstad, P. et al, Breastfeeding, maternal smoking and lower respiratory tract infections. Eur. Respir. J. 1996 / 9 (12) / 2623-2629. , Shu, X.O., et al, Infant breastfeeding and the risk of childhood lymphoma and leukaemia. Int. J. Epidemiol. 1995 / 24 (1) / 27-32. , Wright, A.L. et al, Relationship of infant feeding to recurrent wheezing at age 6 years. Arch. Pediatr. Adolesc. Med. 1995 / 149 (7) / 758-763. , Saarinen, U.M. et al, Breastfeeding as prophylaxis against atopic disease: prospective follow-up study until 17 years old. Lancet 1995 / 346 (8982) / 1065-1069. , Dewey, K.G. et al, Differences in morbidity between breast-fed. And formula-fed infants. J. Pediatr. 1995 / 126 (5 pt 1) / 696-702. , Beaudry, M. et al, Relation between infant feeding and infections during the first six months of life. J. Pediatr. 1995 / 126 (2) / 191-197. , Freudenheim, J.L. et al, Exposure to breastmilk in infancy and the risk of breast cancer. Epidemiology 1994 / 5 (3) / 324-331. , Aniansson, G. et al, A prospective cohort study on breast-feeding and otitis media in Swedish infants. Pediatr. Infect. Dis. J. 1994 / 13 (3) / 183-188. , Verge, C.F. et al, Environmental factors in childhood IDDM. A population-based , case-control study. Diabetes Care 1994 / 17 (12) / 1381-1389. , Mathur, G.P. et al, Breastfeeding and childhood cancer. Indian. Pediatr. 1993 / 30 (5) / 651-657. , Rigas, A. et al, Breast-feeding and maternal smoking in the etiology of Crohn’s disease and ulcerative colitis in childhood. Ann. Epidemiol. 1993 / 3 (4) / 387-392. , Birch, E. et al, Breastfeeding and optimal visual development. J. Pediatr. Ophthalmol. Strabismus. 1993 / 30 (1) / 33-38. , Holberg, C.J. et al, Risk factors for respiratory syncytial virus-associated lower respiratory illnesses in the first yaer of life. Am. J. Epidemiol. 1991 / 133 (11) / 1135-1151. , Popkin, B.M. et al, Breast-feeding and diarrheal morbidity. Pediatrics 1990 / 86 (6) / 874-882. , Lucas, A. et al, Early diet of preterm infants and development of allergic or atopic disease: randomized prospective study. Br. Med. J. 1990 / 300 (6728) / 837-840. , Lucas, A. et al, Breast milk and neonatal necrotizing enterocolitis. Lancet 1990 / 336 (8730) / 519-1523. , Howie, P.W. et al, Protective effect of breast feeding against infection. Br. Med. J. 1990 / 300 (6716) / 11-16. , Wright, A.L. et al, Breast feeding and lower respiratory tract illness in the first year of life. Br. Med. J. 1989 / 299 / (6705) 935-949. , Chen, Y. Synergistic effect of passive smoking and artificial feeding on hospitalisation for respiratory illness in early childhood. Chest. 1989 / 95 (5) / 1004-1007. , Koletzko, S. et al, Role of infant feeding practises in development of Crohn’s disease in childhood. Br. Med. J. 1989 / 298 (6688) / 1617-1618. , Mayer, E.J. et al, Reduced risk of IDDM among breast-fed children. The colorado IDDM Registry. Diabetes 1988 / 37 (12) / 1625-1632. , Davis, M.K. et al, Infant feeding and childhood cancer. Lancet 1988 / 2 (8607) / 365-368. , Cochi, S.L. et al, Primary invasive Haemophilus influenzae type b disease: a population-based assessment of risk factors. J. Pediatr. 1986 / 108 (6) / 887-896. Frank, A.I. et al, Breast feeding and respiratory virus infection. Pediatrics 1982 / 70 (2) / 239-245. , Saarinen, Prolonged breast feeding as prophylaxis for recurrent otitis media. Acta Paediatr. Scand. 1982 / 71 (4) / 567-571.
(4) le Huerou-Luron, I. et al, Source of dietary protein influences kinetics of plasma gut regulatory peptide concentration to feeding in preruminant calves. Comp. Biochem. Physiol. A Mol. Integr. Physiol. 1998 / 119 (3) / 817-824. , Watkins, C.J. et al, The relationship between breast and bottle feeding and respiratory illness in the first year of life. J. Epidemiol. Community Health 1979 / 33 (3) / 180-182.
(5) Takeyama, M. et al, Enzyme immunoassay of gastrin-releasing peptide (GRP)-like immunoreactivity in milk. Int. J. Pept. Protein Res. 1989 / 34 (1) / 70-74.
(6) Ducroc, R. et al, Immunoreactive substance P and calcitonin-gene-related peptide (CGRP) in rat milk and in human milk and infant formula's. Am. J. Clin. Nutr. 1995 / 62 (3) / 554-558.
(7) Faulkner, A. Insuline-like growth factor 1 concentrations in milk and plasma after growth hormone treatment. Biochem. Soc. Trans. 1998 / 26 (4) / 386. , Silanikove, N. et al, Metabolic and productive response of dairy cows to increased ion supplementation at early lactation in warm weather. J. Dairy Res. 1998 / 65 (4) / 529-543. , Ginjala, V. et al, Determination of transforming growth factor-beta 1 (TGF-beta 1) and insulin-like growth factor-1 (IGF-1) in bovine colostreum samples. J. Immunoassay 1998 / 19 (2-3) / 195-207. , Schober, D.A. et al, Perinatal expression of type 1 IGF receptors in porcine small intestine. Endocrinology 1990 / 126 (2) / 1125-1132.
(8) Murphy, M.S. et al, Growth factors and the gastrointestinal tract. Nutrition 1998 / 14 (10) / 771-774. , Buts, J.P. Bioactive factors in milk. (infrench) Arch. Pediatr. 1998 / 5 (3) / 298-306.
(9) Gaull, G.E. et al, Significance of growth modulators in human milk. Pediatrics 1985 / 75 (pt 2) / 142-145.
(10) Hinuma, S. et al, A prolactin-releasing peptide in the brain. Nature 1998 / 393 (6682) / 272-276.
(11) Smith, S.S. et al, Presence of luteinising hormone-releasing hormone (LHRH) in milk. Endocrinol Exp. 1986 / 20 (2-3) / 147-153. , Koldovsky, O. ,Search for the role of milk borne biologically active peptides for the suckling. J.Nutr. 1989 / 119 (11) / 1543-1551.Nair, R.M. et al, Studies on LHRH and physiological fluid amino acids in human colostreum and milk. Endocrinol. Exp. 1987 / 21 (1) / 23-30.
(12) White, M.E. et al, Milk progesterone concentrations following simultaneous administration of buserelin and cloprostenol in cattle with normal corporal lutea. Can. J. Vet. Res. 1986 / 50 (2) / 285-286. , Dinsmore, R.P. et al, Effect of gonadotropin-releasing hormone on clinical response and fertility in cows with cystyic ovaries ,as related to milk progesterone concentration and days after partarition. J. Am. Vet. Med. Assoc. 1989 / 195 (3) / 327-330.
(13) Berseth, C.I. et al, Postpartum changes in pattern of gastrointestinal regulatory peptides in human milk. Am. J. Clin. Nutr. 1990 / 51 (6) / 985-990.
(14) Flood, J.F. et al, Increased food intake by neuropeptide Y is due to an increased motivation to eat. Peptides 1991 / 12 (6) / 1329-1332.
(15) Amarant, T. et al, Luteinising hormone-releasing hormone and thyrotropin-releasing hormone in human and bovine milk. European Journal of Biochemistry 1982 / 127 (3) / 647-650. , Baram ,T. et al, Gonadotropin-releasing hormone in milk. Science 1977 / 198 (4314) / 300-302.
(16) Koike, K. et al, The pituitary folliculo-stellate cell line TtT/GF augments basal and TRH-induced prolactin secretion by GH3 cells. Life Sci. 1997 / 61 (25) / 2491-2497. , Tyson, J.E. et al, The influence of prolactine secretion on human lactation. J. Clin. Endocrinol. Metab. 1975 / 40 (5) / 764-773.
(17) Grochowska, R. et al, Stimulated growth hormone (GH) release in Friesian cattle with respect to GH genotypes. Reprod. Nutr. Dev. 1999 / 39 (2) / 171-180. , Bourne, R.A. et al, Serum growth hormone concentrations after growth hormone or thyroid-releasing hormone in cows. J. Dairy Sci. 1977 / 60 (10) / 1629-1635.
(18) Chomczinsky, P. et al, Stimulatory effect of thyroid hormone on growth hormone gene expression in a human pituitary cell line. J. Clin. Endocrinol. Metab. 1993 / 77 (1) / 281-285. , Reynolds, A.M. ,The effects of chronic exposure to supraphysiological concentrations of 3,5,3`triiodo-L-thyronine (T3) on cultured GC cells. J. Cell. Physiol. 1991 / 149 (3) / 544-547.
(19) Tenore, A. et al, Thyroidal response to peroral TSH in suckling and weaned rats. Am. J. Physiol. 1980 / 238 (5) / E428-430.
(20) Slebodzinski, A.B. et al, Triiodothyronine (T3) ,insulin and characteristics of 5'-monodiodinase (5'-MD) in mare's milk from partarition to 21 days post-partum. Reprod. Nutr. Dev. 1998 / 38 (3) / 235-244.
(21) Fujimoto, N. et al, Upregulation of the estrogen receptor by triiodothyronine in rat pituitary cell lines. J. Steroid. Biochem. Mol. Biol. 1997 / 61 (1-2) / 79-85.
(22) Koldovsky, O., Search for the role of milk borne biologically active peptides for the suckling. J.Nutr. 1989 / 119 (11) / 1543-1551. , Buts, J.P. Bioactive factors in milk. (in french) Arch. Pediatr. 1998 / 5 (3) / 298-306.
(23) Faulkner, A. ,Insulin-like growth factor concentrations in milk and plasma after growth hormone treatment. Biochem. Soc. Trans. 1998 / 26 (4) / S386. , Baldini, E. et al, In vivo cytokinetic effects of recombinant human growth hormone (rhGH) in patients with advanced breast carcinoma. J. Biol. Regul. Homeost. Agents 1994 / 8 (4) / 113-116. , Scheven, B.A. et al, Effects of recombinant human insulin-like growth factor-1 and -2 (IGF) and growth hormone (GH) on the growth of normal adult human osteoblast-like cells and human osteogenic sarcoma cells. Growth Regul. 1991 / 1 (4) / 160-167. , Hodate, K. et al, Plasma growth hormone, insuline-like growth factor-1, and milk production response to exogenous human growth hormone-releasing factor analogs in dairy cows. Endocrinol. Jpn. 1990 / 37 (2) / 261-273.
(24) Westrom, B.R. et al, Levels of immunoreactive insulin, neurotensin, and bombesin in porcine colostreum and milk. J. Pediatr. Gastroenterol. Nutr. 1987 / 6 (3) / 460-465. , Ehman, R. et al, Bombesin, neurotensin and pro-gamma-melanotropin in immunoreactants in human milk. Regul. Pept. 1985 / 10 (2-3) / 99-105.
(25) Shutt, D.A. et al, Comparison of total and free cortisol in bovine serum and milk colostreum. J. Dairy Sci. 1985 / 68 (7) / 1832-1834.
(26) Vaarala, O. et al, Cow milk feeding induces antibodies to insulin in children -- a link between cow milk and insulin-dependent diabetes mellitus ? Scand. J. Immunol. 1998 / 47 (2) / 131-135. , Slebodzinsky, A.B. et al, Triiodothyronine (T3) ,insulin and characteristics of 5'-monodiodinase (5'-MD) in mare's milk from partarition to 21 days post-partum. Reprod. Nutr. Dev. 1998 / 38 (3) / 235-244. , Westrom, B.R. et al, Levels of immunoreactive insulin, neurotensin, and bombesin in porcine colostreum and milk. J. Pediatr. Gastroenterol. Nutr. 1987 / 6 (3) / 460-465.
(27) Ferrando, T. et al, Beta-endorphin-like and alpha-MSH-like immunoreactivities in human milk. Life Sci. 1990 / 47 (7) / 633-635.
(28) Medina, J.H. et al, Presence of benzodiazepine-like molecules in mammalian brain and milk. Biochem. Biophys. Res. Commun. 1988 / 152 (2) / 534-539.
(29) Irvine, C.H. et al, Phytoestrogens in soy-based infant foods : concentrations, daily intake and possible biological effects. Proc. Soc. Exp. Biol. Med. 1998 / 217 (3) / 247-253.
(30) Nagata, C. et al, Effect of soy milk consumption on serum estrogen concentrations in premenopausal Japanese women. J. Natl. Cancer Inst. 1998 / 90 (23) / 1830-1835. , Persky, V. et al, Epidemiology of soy and cancer : perspectives and directions. J. Nutr. 1995 / 125 (3suppl.) / 709S-712S. 
(31) Morris, S.M. et al, p53, mutations, and apoptosis in genistein-exposed human lymphoblastoid cells. Mutat. Res. 1998 / 405 (1) / 41-56. , Kulling, S.E. et al, Induction of micronuclei, DNA strand breaks and HPRT mutations in cultured Chinese hamster V79 cells by the phytoestrogen coumestrol. Food Chem. Toxicol. 1997 / 35 (6) / 605-613.
(32) Stahl, S. et al, Phytoestrogens act as estrogen agonists in an estrogen-responsive pituitary cell line. Toxicol. Appl. Pharmacol. 1998 / 152 (1) / 41-48.
(33) Ducroc, R. et al, Immunoreactive substance P and calcitonin-gene-related peptide (CGRP) in rat milk and in human milk and infant formula's. Am. J. Clin. Nutr. 1995 / 62 (3) / 554-558.
(34) Mandalapu, P. et al, A novel immunosupressiva factor in human colostreum. Cell. Immunol. 1995 / 162 (2) / 178-184.
(35) Okuyama, H. et al, The effect of epidermal growth factor on bacterial translocation in newborn rabbits. J. Pediatr. Surg. 1998 / 33 (2) / 225-228.
(36) Buts, J.P. Bioactive factors in milk. (in french) Arch. Pediatr. 1998 / 5 (3) / 298-306.
(37) Berseth, C.I. et al, Postpartum changes in pattern of gastrointestinal regulatory peptides in human milk. Am. J. Clin. Nutr. 1990 / 51 (6) / 985-990.
(38) Eriksson, U. et al, Insulin-like growth factors (IGF)-1 and -2 and insulin-like growth factor binding proteins (IGFBP) in human colostrum / transitory milk during the first week postpartum : comparison with neonatal and maternal serum. Biochem. Biophys. Res. Commun. 1993 / 196 (1) / 267-273.
(39) Nagashima, K. et al, Levels of insulin-like growth factor-1 in full- and preterm human milk in comparison to levels in cow's milk and in milk formulas. Biol. Neonate 1990 / 58 (6) / 343-346.
(40) Salmenpera, L. et al, Effects of feeding regimen on blood glucose levels and plasma concentrations of pancreatic hormones and gut regulatory peptides at 9 months of age :comparison between infants fed with milk formula and infants exclusively breast-fed from birth. J. Pediatr. Gastroenterol. Nutr. 1998 / 7 (5) / 651-656.
(41) Halliwell, B. and M.C. Gutteridge, Role of free radicals and catalytic metal ions in human disease : An overview. Methods Enzymol. 186 (1990) / 1-85. , Kehrer, J.P., Free radicals as mediators of tissue injury and disease. Crit.Reviews Toxicol.23 (1993) / 21-48. , Yu, B.P., Cellular defenses against damage from reactive oxygen species. Physiol. Reviews 74 (1994) / 139-161.
(42) Lee, T.S. et al, Iron-deficient diet reduces atherosclerosis lesions in ApoE-deficient mice Circuation 1999 / 99 (9) / 1222-1229. , Patel, R.P. et al, Formation of oxysterols during oxidation of low density lipoprotein by peroxynitrite, myoglobine, and copper. J. Lipid. Res. 1996 / 37 (11) / 2361-2371. , Dzeletovic, S. et al, Time course of oxysterol formation during in vitro oxidation of low density lipoprotein. Chem. Phys. Lipids 1995 / 78 (2) / 119-128. , Herbert, V. et al, Iron worsenes high cholesterol-related coronary artery disease. Am. J. Clin. Nutr. 1994 / 60 (2) / 299-300.
(43) Oikawa, S. et al, Distinct mechanisms of site-specific DNA damage induced by endogenous reductants in the presence of iron (III) and copper (II). Biochem. Biophys. Acta 1998 / 1399 (1) / 19-30.
(44) Sok, D.E., Oxidative inactivation of brain alkaline phosphatase responsible for hydrolysis of phosphocholine. J. Neurochem. 1999 / 72 (1) / 355-362.
(45) Burke, W. et al, Hemachromatosis : genetics helps to define a multifactorial disease. Clin. Genet. 1998 / 54 (1) / 1-9. , Crawford, R.D., Proposed role for a combination of citric acid and ascorbic acid in the production of dietary iron overload : a fundamental cause of disease Bichem. Mol. Med. 1995 / 54 (1) / 1-11. , Britton, R.S. et al, Pathophysiology of iron toxicity. Adv. Exp. Med. Biol. 1994 / 356 / 239-253. , Phatak, P.D. et al, Management of hereditary hemachromotosis. Blood Rev. 1994 / 8 (4) / 193-198.
(46) Sawa, T. et al, Lipid peroxyl radicals from oxidized oils and heme-iron :implication of a high fat diet in colon carcinogenesis. Cancer Epidemiol. Biomarkers Rev. 1998 / 7 (11) / 1007-1012.
(47) Jellinger, K.A., The role of iron in neurodegeneration : prospects for pharmacotherapy of Parkinson's disease. Drugs. Aging. 1999 / 14 (2) / 115-140. , Spencer, J.P. et al, Conjugates of catecholamines with cysteine and GSH in Parkinson's disease : possible mechanisms of formation involving reactive oxygen species. J. Neurochem. 1998 / 71 (5) / 2112-2122. , Snyder, R.D. et al, Enhancement of cytotoxicity and and clastogenicity of L-dopa and dopamine by manganese and copper. Mutat. Res. 1998 / 405 (1) / 1-8. , Vescovi, A. et al, Interactions of manganes with human brain glutathione-S-transferase. Toxicology 1989 / 57 (2) / 183-191.
(48) Olsen, P.A. et al, Effects of supplementation of organic and inorganic combinations of copper, cobalt, manganese, and zinc above nutrient requirement levels on postpartum two-year-old cows. J. Anim. Sci. 1999 / 77 (3) / 522-532.
(49) Bermejo, P. et al, Zinc Specification in Protein Fractions of Different Kinds of Milk by HPLC-FAAS. in : Köhrle, J., Mineralstoffe und Spuren-elemente, Wissenschaftliche Verlagsgesellschaft mbH Stuttgart 1998 / 131-136 en 143-147. , Wigertz, K. et al, Effect of milk processing on the concentration of folate-binding protein (FBP) ,folate binding capacity and retention of S-methyltetrahydrofolate. Int. J. Food Sci. Nutr. 1996 / 47 (4) / 315-322. , Lonnerdal, B., Bioavailibility of copper. Am. J. Clin. Nutr. 1996 / 63 (5) / 821S-829S. , Lonnerdal, B. ,Dietary factors affecting trace element bioavailability from human milk, cow's milk, and infant formulas. Prog. Food Nutr. Sci. 1985 / 9 (1-2) / 35-62. , Lonnerdal, B. et al, Manganese binding proteins in human and cow's milk. Am. J. Clin. Nutr. 1985 / 41 (3) / 550-559. , Lönnerdal, B. et al, The effect of individual components of soy formula and cow's milk formula on zinc bioavailability. Am. J. Clin. Nutr. 1984 / 40 (5) / 1064-1070. , Craig, W.J. et al, Zinc bioavailability and infant formulas. Am. J. Clin. Nutr. 1984 / 39 / 981-983. , Harzer, G. et al, Binding of zinc to casein. Am. J. Clin. Nutr. 1982 / 35 (5) / 981-987. , Bermejo, P. et al ,Study of Copper Specification in Milks used in Lactation of Human Infants by HPLC-ETAAS.
(50) Divi, R.L. et al, Anti-thyroid isoflavones from soyabean, isolation, characterization, and mechanisms of action. Biochemical Pharmacology 1997 / 54 (10) / 1087-1096.
(51) Souci, S.W. et al, Food Composition and Nutrition Tabels. Medpharm Scientific Publishers Stuttgart 1994 / 6-28.
(52) Bunin, G.R. ,Maternal diet during pregnancy and risk of brain tumors in children. Int. J. Cancer Suppl. 1998 / 11 / 23-25. , Mc Bride, M.L. et al, Childhood cancer and environmental contaminants. Can. J. Public. Health 1998 / 89 / suppl.1 / S53-62, S58-68. , Preston-Martin, S. et al, Maternal consumption of cured meats and vitamins related to pediatric brain tumors. Cancer Epidemiol. Biomarkers Prev. 1996 / 5 (8) / 599-605. , Mirvish, S.S. ,Role of N-nitroso compounds (NOC) and N-nitrosation in etiology of gastric, esophageal, nasopharyngeal and bladder cancer and contribution to cancer of known exposures to NOC. Cancer Lett. 1995 / 93 (1) / 17-48. , Bunin, G.R. et al, Maternal diet and risk of astrocytic glioma in children : a report from the Childrens Cancer Group. Cancer Causes Control 1994 / 5 (2) / 177-187. , Sarasna, S. et al, Cured and broiled meat consumption in relation to childhood cancer : Denver ,Colorado. Cancer Causes Control 1994 / 5 (2) / 141-148. , Kuijten, R.R. et al, Gestational and familial risk factors for childhood astrocytoma : results of a case control study. Cancer Res. 1990 / 50 (9) / 2608-2612. , Preston-Martin, S. et al, N-nitroso compounds and childhood brain tumors : a case control study. Cancer Res. 1982 / 42 (12) / 5240-5245.
(53) Sumihara, K. ,Recent problem of trans-fatty acids in human milk (Japans). Nikon Kango Kangakkaishi 1997 / 17 (1) / 58-65. , Chen, Z.Y. ,Breast milk fatty acid composition : a comparative study between Hong Kong and Chongqing Chinese. Lipids 1997 / 32 (10) / 1061-1067. , Ratnayake ,W.M. et al, Trans, n-3, and n-6 fatty acids in Canadian human milk. Lipids 1996 / 1996 / 31 suppl./ S279--282. , Chardigny J.M. et al, Trans mono- and polyunsaturated fatty acids in human milk. Eur. J.Clin. Nutr. 1995 / 49 (7) / 523-531.
(54) Pietinen, P. et al, Intake of fatty acids and risk of coronary heart disease in a cohort of Finish men. The Alpha-Tocopherol, Beta-Carotene Cancer Prevention Study. Am. J. Epidemiol. 1997 / 145 (10) / 876-887. , Zock, P.L. et al, Trans-fatty acids ,lipoproteins, and coronary risk. Can. J. Physiol. Pharmacol. 1997 / 75 (3) / 211-216. , Singh, R.B. et al, Association of trans-fatty acids (vegetable ghee) and clarified butter (Indian ghee) intake with higher risk of coronary artery disease in rural and urban populations with low fat consumption. Int. J. Cardiol. 1996 / 56 (3) / 289-298 / disc. 299-300. , Hodgson, J.M. et al, Platelet trans fatty acids in relation to angiographically assessed coronary artery disease. Atherosclerosis 1996 / 120 (1-2) / 147-154. , Temple, N.J. Dietary fats and coronary heart disease. Biomed. Pharmacother. 1996 / 50 (6-7) / 261-268. , Watts, G.F. et al, Relationship between nutrient intake and progression / regression of coronary atherosclerosis as assessed by serial quantative angiography. Can. J. Cardiol. 1995 / 11 / suppl. G / 110G-114G. , Stender, S. et al, The influence of trans-fatty acids on health : a report from the Danish Nutrition Council. Clin. Sci. (Colch.) 1995 / 88 (4) / 375-392. , Willet, W.C. et al, Trans-fatty acids : are the effects only marginal ? Am. J. Public Health 1994 / 84 (5) / 722-724. , Ascherio, A. et al, Trans-fatty acid intake and risk of myocardial infarction. Circulation 1994 / 89 (1) / 94-101. , Siguel, E.N., Trans fatty acid patterns in patients with angiographically documented coronary artery disease. Am. J. Cardiol. 1993 / 71 (11) / 916-920. , Willet, W.C. et al, Intake of trans fatty acids and risk of coronary heart disease among women. Lancet 1993 / 341 (8845) / 581-585.
(55) Kohlmeier, L. et al, Adipose tissue trans-fatty acids and breast cancer in the European Community Multicenter Study on Antioxidants, Myocardial Infarction, and Breast Cancer. Cancer Epidemiol. Biomarkers Prev. 1997 / 6 (9) / 705-710.
(56) Williams, M.A. et al, Risk of preeclampsia in relation to elaidic acid (trans-fatty acid) in maternal erythocytes. Gynecol. Obstet. Invest. 1998 / 46 (2) / 84-87.
(57) Fukushima, Y. et al, Consumption of cow milk and egg by lactating women and the presence of beta-lactoglobulin and ovalbumin in breast milk. American Journal of Clinical Nutrition 1997 / 65 (1) / 30-35.
(58) Mackey, A.D. et al, Maternal folate status during extended lactation and the effect of supplemental folic acid. Am. J. Clin. Nutr. 1999 / 69 (2) / 285-292.
(59) Moos, F.C. et al, GABA-induced facilitation of the periodic bursting activity of oxytocin neurones in suckled rats. J.Physiol. (Lond.) 1995 / 488 (Pt1) / 103-114. , Voisin, D.L. et al, Central inhibitory effects of muscinol and bicuculline on the milk ejection reflex in the anaesthetized rat. J. Physiol. (Lond.) 1995 / 483 (Pt1) / 211-224.
(60) Donnerer, J. ,Evidence for an excitatory action of the benzodiazepine receptor inverse agonist FG7142 on C-fibre afferents. Naunyn Schmiedebergs Arch. Pharmacol. 1989 / 340 (3) / 352-354. , Insel, T.R. et al, Rearing paradigm in a nonhuman primate affects response to beta-CCE challenge. Psychopharmacology (Berl.) 1988 / 96 (1) / 81-86. , Glowa, J.R. et al, Effects of beta-carboline-3-carboxylic acid ethyl ester on suppressed and non-suppressed responding in the rhesus monkey. Eur. J. Pharmacol. 1986 / 129 (1-2) / 39-47. , Insel, T.R. et al, A benzodiazepine receptor-mediated model of anxiety studies in nonhuman primates and clinical implications. Arch. Gen. Psychiatry 1984 / 41 (8) / 741-750. , Ninan, P.T. et al, Benzodiazepine-mediated experimental ''anxiety'' in primates. Science 1982 / 218 (4579) / 1332-1334.
(61) Jarvinen, A. et al, Effects of central and peripheral type benzodiazepine ligands on thyrotropin and prolactin secretion. Neuropeptides. 1992 / 21 (3) / 183-191. , Jarvinen, A. et al, Central and peripheral type benzodiazepine ligands displace (3H)(3-Me-His2)TRH from its binding sites in the brain and the anterior pituitary and antagonize the effect of TRH in the rat duodenum. Neuropeptides 1991 / 19 (3) / 147-155. , De Deyn, P.P. et al, Epilepsy and the GABA-hypothesis ,a brief revieuw and some examples. Acta. Neurol. Belg. 1990 / 90 (2) / 65-81. , Roussel, J.P. et al, Benzodiazepines inhibit thyrotropin (TSH)-releasing hormone-induced TSH and growth hormone release from perifused rat pituitaries. Endocrinology 1986 / 119 (6) / 2519-2526. , Smythe, G.A. et al, Effects of 6-methoxy-1,2,3,4-tetrahydro-beta-carbolin and yohimbine on hypothalamic monoamine status and pituitary hormone release in the rat. Aust. J. Biol. Sci. 1983 / 36 (4) / 379-386.
(62) Kowalsky, W.B. et al, Morphine inhibits nocturnal oxytocin secretion and uterine contractions in the pregnant baboon. Biol. Reprod. 1998 / 58 (4) / 971-976.
(63) Lindow, S.W. et al, Morphine suppresses the oxytocin response in breast-feeding women. Gynecol. Obstet. Invest. 1999 / 48 (1) / 33-37.
(64) Suda, T. et al, Beta-endorphin inhibits hypoglycemia-induced gene expression of corticotropin-releasing factor in the rat hypothalamus. Endocrinology 1992 / 130 (3) / 1325-1330
(65) Chow, S.Y. et al, Brain oxytocin receptor antagonism disinhibits sodium appetite in preweanling rats. Regul. Pept. 1997 / 68 (2) / 119-124. , Arletti, R. et al, Influence of oxytocin on feeding behavior in the rat. Peptides 1989 / 10 (1) / 89-93.
(66) Chua, S. et al, Influence of breastfeeding and nipple stimulation on postpartum uterine activity. Br. J. Obstet. Gynaecol. 1994 / 101 (9) / 804-805.
(67) Moos, F.C. et al, Electrical recordings of magnocellular supraoptic and paraventricular neurons displaying both oxytocine- and vasopressin-related activity. Brain Res. 1995 / 669 (2) / 309-314.
(68) Lindow, S.W. et al, Morphine suppresses the oxytocin response in breast-feeding women. Gynecol. Obstet. Invest. 1999 / 48 (1) / 33-37.
(69) Bing, C. et al, The effect of moxonidine on feeding and body fat in obese Zucker rats : role of hypothalamic NPY neurones. Br. J. Pharmacol. 1999 / 127 (1) / 35-42. , Asakawa, A. et al, Urocortin reduces food intake and gastric emptying in lean and ob/ob obese mice. Gastroenterology 1999 / 116 (6) / 1287-1292. , Nishiyama, M. et al, Leptin effects on the expression of type-2 CRH receptor mRNA in the ventromedical hypothalamus in the rat. J. Endocrinol. 1999 / 11 (4) / 307-314. , Hakansson, M.L. et al, Leptin receptor immunoreactivity in chemically defined target neurons of the hypothalamus.J. Neurosci. 1998 / 18 (1) / 559-572. , Plamondon, H. et al, Anorectic action of bombesin requires receptor for corticotropin-releasing factor but not for oxytocin. Eur. J. Pharmacol. 1997 / 340 (2-3) / 99-109.
(70) Brogan, R.S. et al, Suppression of leptin during lactation : contribution of the suckling stimulus versus milk production. Endocrinology 1999 / 140 (6) / 2621-2627.
(71) Fortun-Lamothe, L. et al, Influence of prolactin on in vivo and in vitro lipolysis in rabbits. Comp. Biochem. Physiol. C. Pharmacol. Toxicol. Endocrinol. 1996 / 115 (2) / 141-147.
(72) Dewey, K.G. et al, Maternal weight loss patterns during prolonged lactation. Am. J. Clin. Nutr. 1993 / 58 (2) / 162-166.
(73) Tay, C.C. et al, Effect of antagonists of dopamine and opiates on the basal and GnRH-induced secretion of Luteinizing hormone, Follicel-stimulating hormone and Prolactin during lactational amenorrhea in breastfeeding women. Hum. Reprod. 1993 / 8 (4) / 532-539.
(74) Vekemans, M. ,Postpartum contraception : the lactational amenorrhea method. Eur. J. Contracept. Reprod. Health Care 1997 / 2 (2) / 105-111. , Campbell, O.M. et al, Characteristics and determinants of postpartum ovariuan function in women in the United States. Am. J. Obstet. Gynecol. 1993 / 169 (1) / 55-60. , Kennedy, K.I. et al, Contraceptive efficacy of lactational amenorrhoea. Lancet 1992 / 339 (8787) / 227-230. , Labbock, M.H. et al, Puerperium and breast-feeding. Curr. Opin. Obstet. Gynecol. 1992 / 4 (6) / 818-825.
(75) Lipworth, L. et al, History of breast-feeding in relation to breast cancer risk : a revieuw of the epidemiologic literature. J. Natl. Cancer Inst. 2000 / 92 (4) / 302-312. , Potischman, N. et al, In-utero and early life exposures in relation to risk of breast cancer. Cancer Causes Control 1999 / 10 (6) / 561-573. , Zhao, Y. et al, Matched case-control study for detecting risk factors of breast cancer in women living in Chengdu. (in Chinese) Chung Hua Liu Hsing Ping Hsueh Tsa Chih 1999 / 20 (2) / 91-94. , Newcomb, P.A. et al, Lactation in relation to postmenopausal breast cancer. Am. J. Epidemiol. 1999 / 150 (2) / 174-182. , Egan, K.M. et al, Risk factors for breast cancer in women with a breast cancer family history. Cancer Epidemiol. Biomarkers Prev. 1998 / 7 (5) / 359-364. , McCredie, M. et al, Reproductive factors and breast cancer in New Zealand. Int. J. Cancer 1998 / 76 (2) / 182-188. , Newcomb, P.A. ,Lactation and breast cancer risk. J. Mammary Gland Biol. Neoplasia 1997 / 2 (3) / 311-318. , Newcomb, P.A. et al, Lactation and a reduced risk of premenopausal breast cancer. N. Engl. J. Med. 1994 / 330 (2) / 81-87.
(76) Rosenblatt, K.A. et al, Lactation and the risk of epithelial ovarian cancer. The WHO Collaberative Study of Neoplasia and Steroid Contraceptives. Int. J. Epidemiol. 1993 / 22 (2) / 192-197. , Schneider, A.P. 2d, Risk factor for ovarian cancer. N. Engl. J. Med. 1987 / 317 (8) / 508-509.
(77) Rosenblatt, K.A. et al, Prolonged lactation and endometrial cancer. WHO Collaborative Study of Neoplasia and Steroid Contraceptives. Int. J. Epidemiol. 1995 / 24 (3) / 499-503.
(78) Minda H, et al, Effect of different types of feeding on fatty acid composition of erythrocyte membrane lipids in full-term infants. Acta Paediatr 2002;91(8):874-81.
(79) Cheruku SR, et al, Higher maternal plasma docosahexaenoic acid during pregnancy is associated with more mature neonatal sleep-state patterning. Am J Clin Nutr 2002 Sep;76(3):608-13.